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One major hurdle in drug development is an incomplete understanding of the underlying mechanisms that drive Aβ plaques and tau-containing neurofibrillary tangles (NFTs) in the brain which may begin decades prior to cognitive decline (9-11)
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Research using high-glucose-injured human retinal pigment epithelial cells (ARPE-19) showed Epithalon treatment restored impaired wound healing, reduced intracellular reactive oxygen species accumulation, and increased antioxidant gene expression
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